Renin-angiotensin-aldosteron system (RAAS) make vasoconstriction and reabsorption of water and sodium in compensation stage of shock.
Shock is a condition where there is an imbalance between the supply and demand of oxygen in the body. In the initial phase, the state of shock can be compensated by the body (compensation stage) such as by increasing the pulse, redistributing blood to vital organs, and so on so that blood pressure can still be measured normally. One of the systems that play a role in maintaining blood pressure is the Renin-Angiotensin-Aldosterone System (RAAS).
When hypovolemia (decreased intravascular volume) or hypotension (low blood pressure) occurs, the baroreceptors detect it. It also occurs hypoperfusion to the renal tissue. This causes it to be detected in the baroreceptors on the afferent arterioles.
Signals from these baroreceptors will increase renin, so that angiotensinogen changes to angiotensin I. Then angiotensin I will change to angiotensin II by the angiotensin converting enzyme.
This will result in:
These three mechanisms will cause an increase in blood pressure.
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